The human brain is mainly composed of neurons, neuroglia, and brain tracts, consisting of tens of thousands of nerve cells. White matter belongs to the central nervous system tract, and leukoaraiosis refers to the chronic ischemic degenerative process that begins when sympathetic nerves reach the white matter part due to various reasons such as trauma, poisoning, vascular disease, systemic diseases, and nutritional deficiencies, leading to the gradual necrosis and apoptosis of white matter cells, the gradual decline of original function, and the manifestation of dizziness, decreased memory, memory impairment, or decreased function of some parts of the central nervous system, even ataxia, dementia, and so on.

In a strict sense, leukoaraiosis is not a single disease. Its essence is the manifestation of degenerative changes in brain function in imaging diagnosis, pathological changes are white matter demyelination, also known as leukoaraiosis. According to the size of the lesion, imaging diagnosis can be divided into 1-4 grades, among which grade 1 is the mildest and grade 4 is the most severe. In cranial magnetic resonance imaging, the presence of patchy high-density shadows around the lateral ventricles and in the centrum semiovale can be described as leukoaraiosis under the condition of excluding new cerebral infarction. Accompanying leukoaraiosis are brain atrophy, brain sulci, and ventricular thickening.

In the short term, patients with leukoaraiosis do not have any discomfort. With the passage of time, the expansion of the total area of leukoaraiosis results in significant degenerative changes in brain function. Patients may experience non-specific clinical manifestations such as dizziness, decreased memory, and memory impairment, which may eventually develop into cognitive impairment and dementia.

Therefore, when leukoaraiosis reaches grade 2, it is necessary to carry out active intervention, such as thinking more, pondering more, watching less TV, and taking drugs that can moderately nourish the brain. These drugs can also promote brain metabolism, such as nimodipine, oxiracetam, and sodium cytidine. Naturally, this medication must be carried out under the specific guidance of a physician.