Experts introduce that there are many reasons for low platelets, common ones include hereditary, acquired, non-immune, and immune. Once the disease is detected, it must be treated actively. In addition, attention should be paid to diet, eating more foods that can promote platelet production, such as peaches can also be eaten.

Patients with monocytic leukemia can eat peaches.

Peach; known as ‘Shou Peach’ and ‘Xian Peach’ for its delicious flesh, it is also called ‘the number one fruit in the world’. Peach flesh contains protein, fat, carbohydrates, crude fiber, calcium, phosphorus, iron, carotene, vitamin B1, and organic acids (mainly malic acid and citric acid), sugars (mainly glucose, fructose, sucrose, xylose), and volatile oil. Peaches are suitable for patients with hypokalemia and iron deficiency anemia.

1. Decreased platelet production

(1) Genetic: such as Fanconi anemia, congenital thrombocytopenia without megakaryocytes, and May-Hegglin anomaly, etc.

(2) Acquired: aplastic anemia, bone marrow infiltration (such as bone marrow metastasis of malignant tumors, leukemia, bone fibrosis, tuberculosis), chemotherapy drugs, radiation, megakaryocytic aplasia, viral infection (measles, epidemic parotitis), drugs affecting platelet production (such as alcohol), vitamin B and folic acid deficiency.

2. Increased platelet destruction caused by non-immune factors

Thrombotic thrombocytopenic purpura, pregnancy, infection, vascular tumor-platelet syndrome, snake bite, acute respiratory distress syndrome, severe burns, etc.

3. Increased platelet destruction caused by immune factors

Immune thrombocytopenic purpura, HIV infection, cyclic thrombocytopenia, thrombocytopenia caused by drugs (such as heparin, quinine, quinidine, antipyretic and analgesics, penicillin, cephalosporin antibiotics, rifampicin, furosemide, carbamazepine, sodium valproate, sulfonylurea hypoglycemic agents, and phenytoin sodium), and thrombocytopenia after blood transfusion.

4. Excessive retention of platelets in the spleen

Hyperplasia of the spleen leads to a retention effect on platelets by the spleen, resulting in a decrease in the number of platelets in the blood. For example, Gaucher’s disease often leads to hyperplasia of the spleen, and patients may have a tendency to bleed due to thrombocytopenia caused by hyperplasia of the spleen, commonly manifested as fatigue, easy bruising, and nosebleeds. In the ICGG report, 56% of Gaucher patients worldwide will experience thrombocytopenia symptoms. Some children may also have anemia, which may be mainly caused by the shortening of red blood cell lifespan due to hyperplasia of the spleen. Gaucher’s disease is an autosomal recessive inherited disease caused by a mutation in the glucocerebrosidase gene, resulting in a lack of activity of the glucocerebrosidase enzyme in the body, causing its substrate glucocerebroside to accumulate in the lysosomes of macrophages in the liver, spleen, bones, lungs, and even brain. The clinical manifestations include involvement of multiple organs, progressive, and can be life-threatening. Patients can be diagnosed by detecting the activity of the glucocerebrosidase enzyme.